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Tick Boot Camp

Tick Boot Camp

Von: Matt Sabatello and Rich Johannesen
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 The goal of the Tick Boot Camp Podcast is to help people liberate themselves and others from suffering caused by Lyme disease through validation, community building, belief that healing is possible, and modeling success. Listen to our Tick Boot Camp podcast using all major podcast streaming services such as Apple Podcasts, Spotify, and YouTube Music. Our podcast is also integrated with smart home devices, such as Amazon Alexa and Apple TV. Ask your device to "play the Tick Boot Camp Podcast!"Copyright 2025 Alternative & Komplementäre Medizin Hygiene & gesundes Leben
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  • Episode 549: How Chronic Illness Really Works: Dr. Eric Gordon on Lyme, Mold, MCAS, and the Path Back to Health — Tick Boot Camp

    Episode 549: How Chronic Illness Really Works: Dr. Eric Gordon on Lyme, Mold, MCAS, and the Path Back to Health — Tick Boot Camp
    Jan 10 2026
    In this episode of the Tick Boot Camp Podcast, Dr. Eric D. Gordon — globally recognized expert in Lyme disease, ME/CFS, mold toxicity, MCAS, mitochondrial dysfunction, and complex chronic illness — explains why chronic illness is never caused by a single factor and why recovery requires a strategic “order of operations.” Recorded after meeting at Project Lab Coat during NYFW, this conversation dives into chronic inflammation, immune dysregulation, why some people stay sick for years, why certain treatments backfire, how metabolomics reveals dysfunction that standard tests miss, and the future of individualized chronic illness care. Guest Bio Medical Director, Gordon Medical Associates, and President, Gordon Medical Research Center Dr. Gordon has 45+ years of experience treating the most complex chronic illness cases. He specializes in: Lyme disease and tick-borne infections ME/CFS and post-infectious illness Mold and mycotoxin exposure Mast Cell Activation Syndrome (MCAS) Autoimmune disease Environmental illness Mitochondrial dysfunction and metabolic collapse He co-authored the landmark 2016 PNAS metabolomics study with Dr. Robert Naviaux, which reshaped global understanding of ME/CFS and chronic inflammatory diseases. Key Topics Covered How Dr. Gordon became one of the world’s leading chronic illness clinicians Why patient belief and validation are foundational to healing Why chronic illness cases don’t fit conventional medical models Why herbs often worsen symptoms in MCAS or inflamed patients When pharmaceuticals help stabilize sensitive patients How chronic inflammation blocks trace mineral absorption The link between minerals, B vitamins, mitochondria, and NAD/NADH When detoxification helps — and when it causes more harm How childhood infections and environment shape lifelong immunity The massive impact of modern microbiome disruption Mold illness as the “great derailer” of Lyme treatment Why genetics like MTHFR and HLA are not destiny Why some people heal from Lyme without treatment How metabolomics and AI will usher in precision medicine What actually keeps people sick — accumulated compensations, not the tick bite What intuitive patients get right (and wrong) about their symptoms Timestamps 0:02 – Meeting Dr. Gordon at Project Labcoat 1:08 – Who he is and how he entered complex illness medicine 2:30 – Realizing conventional medicine fails chronic patients 5:45 – Why chronic illness doesn’t fit standard algorithms 8:10 – Herbs vs antibiotics: what most people misunderstand 11:28 – Inflammation and why sensitive patients react to everything 13:45 – MCAS and immune overactivation 16:25 – Why herbal formulas can trigger flares 19:30 – Pharmaceuticals that calm inflammation 20:50 – Trace minerals, mitochondrial function, and NAD pathways 23:55 – Why standard labs can’t see cellular dysfunction 26:10 – How childhood immune experiences shape resilience 28:40 – Environmental changes and microbiome decline 30:30 – Shoes, posture, fascia, lymphatics 36:35 – Structural healing and hypersensitive patients 41:20 – Founding Gordon Medical Associates 43:00 – Early discoveries with Lyme disease patients 48:30 – Detoxification, herbal protocols, and mold models 52:10 – Mold’s ability to halt all progress 55:30 – Why mold affects some family members and not others 57:20 – How food supply antibiotics disrupt immunity 59:50 – Genetics are possibilities, not fate 1:03:20 – Why some people recover after a tick bite and others don’t 1:07:00 – How AI and metabolomics will transform treatment 1:10:40 – Genes vs environment 1:13:30 – Chronic illness requires many small steps 1:16:00 – How to work with Dr. Gordon 1:18:30 – Final message of hope Pull Quotes “Chronic illness is not caused by one thing — and it’s never healed by one thing.” “Herbs depend on your body’s ability to modulate inflammation. If you can’t dampen the fire, herbs feel like gasoline.” “Genetics are not destiny. They’re possibilities.” “Mold makes every other treatment look like it’s failing.” “You can absolutely get well — but there is no single magic bullet.” Call to Action If this episode brought you clarity or hope, please share it with someone navigating chronic Lyme, mold illness, MCAS, or ME/CFS. Subscribe and leave a review to help more people find this conversation and believe that healing is possible.
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    1 Std. und 20 Min.
  • Episode 548: When Infections Leave a Mark: How the Immune System Remembers and Shapes Alzheimer’s | Dr. Betsy Bradshaw

    Episode 548: When Infections Leave a Mark: How the Immune System Remembers and Shapes Alzheimer’s | Dr. Betsy Bradshaw
    Dec 16 2025
    Overview This special episode of the [Tick Boot Camp Podcast](https://tickbootcamp.com/podcast/) was recorded live at the 2nd Annual Alzheimer’s Pathobiome Initiative (AlzPI) and PCOM Symposium in collaboration with Pathobiome Perspectives. Hosted by Ali Moresco in partnership with Nikki Schultek, Executive Director of AlzPI, the conversation continues the Tick Boot Camp mission of exploring infection-associated chronic illness (IACI)—including Lyme disease and other tick-borne infections—within the global Alzheimer’s and neuroimmunology research community. Tick Boot Camp co-founders Matt Sabatello and Rich Johannesen partnered with Ali and Nikki to highlight leading scientists connecting microbes, immune dysregulation, and neurodegenerative disease. This episode features Dr. Elizabeth “Betsy” Bradshaw, Assistant Professor of Neurology at Columbia University Irving Medical Center, whose research investigates how past infections leave lasting imprints on the brain’s immune system and influence the development of Alzheimer’s disease. Guest Elizabeth M. Bradshaw, PhD Assistant Professor of Neurology, Columbia University Irving Medical Center Principal Investigator, Bradshaw Laboratory – Neuroimmunology and Genetics of Alzheimer’s Dr. Bradshaw’s laboratory focuses on the immune system’s role in neurodegeneration, particularly how infection and inflammation alter brain immunity and predispose individuals to conditions like Alzheimer’s disease. Her work builds on large-scale genome-wide association studies (GWAS) that identified immune-related genetic variants linked to Alzheimer’s susceptibility, suggesting that subtle changes in immune function—not just neuronal factors—may underlie disease onset. Her team is exploring how pathogens such as HSV-1 (Herpes Simplex Virus Type 1) interact with the brain’s immune cells, known as microglia, and how these infections can “reprogram” immune responses long after the pathogen is cleared. Key Discussion Points Dr. Bradshaw explains how her research bridges genetics, immunology, and infectious disease to better understand Alzheimer’s. Through GWAS data, her team found that many of the genes linked to Alzheimer’s risk involve immune pathways rather than neuronal signaling. This discovery redirected the field’s attention toward how immune cells respond to pathogens and environmental stressors across a lifetime. Using human-derived microglia-like cells created from blood monocytes, her team observes how infections reshape immune cell metabolism and memory. By infecting these microglia-like cells with Alzheimer’s-associated pathogens like HSV-1, they study how genetic background and infection history determine immune cell behavior. The findings suggest that past infections may epigenetically and metabolically train microglia—changing how they respond to aging, stress, and amyloid buildup. Even when the infection has resolved, these “reprogrammed” immune cells can remain altered for decades, silently increasing the brain’s vulnerability to neurodegeneration. Dr. Bradshaw emphasizes that understanding how infections rewire the brain’s immune landscape could transform early intervention strategies. Identifying combinations of genetic risk factors and pathogen exposures may enable targeted prevention or immune-modulating treatments long before symptoms appear. “Microglia remember. Even after the pathogen is gone, they carry its imprint—responding differently decades later when the brain faces new challenges.” — Dr. Elizabeth Bradshaw Why It Matters Dr. Bradshaw’s work reframes Alzheimer’s disease as a neuroimmune condition shaped by infection and host genetics. Her research highlights how microbial exposures, immune history, and inflammation converge to influence cognitive decline. By integrating infection biology with genetics and immunology, her team is redefining how scientists and clinicians view the root causes of Alzheimer’s and other neurodegenerative diseases. This work strengthens the growing case that the immune system’s “memory” of infection may be one of the most important and overlooked factors in brain health and aging. About the Event This interview was recorded at the 2nd Annual Alzheimer’s Pathobiome Initiative (AlzPI) and Philadelphia College of Osteopathic Medicine (PCOM) Symposium, held October 3, 2025, at Ohio University in Dublin, Ohio. The event brought together more than 20 leading researchers exploring how microbes, the microbiome, and immune dysregulation contribute to Alzheimer’s, dementia, and infection-associated chronic illness (IACI). Tick Boot Camp partnered with Ali Moresco and Nikki Schultek to share these conversations and connect chronic Lyme, infection, and neurodegenerative research communities. Learn More Learn more about the Alzheimer’s Pathobiome Initiative (AlzPI) Listen to Tick Boot Camp Podcast episodes, including Episode 406: Pathobiome – An ...
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    18 Min.
  • Episode 547: How Bartonella Hijacks the Brain’s Immune System: Linking Infection and Neurodegeneration – Dr. Janice Bush

    Episode 547: How Bartonella Hijacks the Brain’s Immune System: Linking Infection and Neurodegeneration – Dr. Janice Bush
    Dec 9 2025
    Overview This special episode of the Tick Boot Camp Podcast was recorded live at the 2nd Annual Alzheimer’s Pathobiome Initiative (AlzPI) and PCOM Symposium in collaboration with Pathobiome Perspectives. Hosted by Ali Moresco in partnership with Nikki Schultek, Executive Director of AlzPI, this series expands the Tick Boot Camp mission of exploring infection-associated chronic illness (IACI)—including Lyme and other tick-borne infections—to the global Alzheimer’s and neuroimmunology research community. Tick Boot Camp co-founders Matt Sabatello and Rich Johannesen partnered with Ali and Nikki to showcase scientists exploring the microbial and immune mechanisms behind neurodegeneration. This episode features Dr. Janice Bush, a PhD candidate at North Carolina State University’s College of Veterinary Medicine, whose research under world-renowned Bartonella expert Dr. Edward Breitschwerdt investigates how Bartonella bacteria alter gene expression in the brain’s immune cells. Guest Janice Bush, DVM, PhD CandidateCollege of Veterinary Medicine, North Carolina State University (NCSU) Dr. Janice Bush began her career in veterinary medicine, where she observed a striking overlap between illnesses in pets and their human owners—particularly those linked to vector-borne infections like Bartonella. Now completing her PhD under Dr. Edward Breitschwerdt, she focuses on Bartonella henselae, the bacterium behind Cat Scratch Disease, and its ability to infect human microglial cells—the brain’s resident immune defenders. Her presentation, “Bartonella-Infected Human Microglial Cells: Transcriptional Changes Associated with Chronic Neurologic Disorders,” revealed how this stealth pathogen triggers widespread gene dysregulation linked to Alzheimer’s disease, psychiatric symptoms, and neurodegenerative processes. Key Discussion Points Dr. Bush explains how Bartonella infection reprograms human microglia, the brain’s innate immune cells, leading to hundreds of genes being upregulated or suppressed—affecting energy metabolism, mitochondrial function, cell signaling, and immune communication. These cellular changes mirror those observed in chronic neurological and psychiatric disorders, providing a potential mechanistic link between infection and long-term neurodegeneration. She describes Bartonella’s sophisticated immune evasion strategy, including its ability to hijack cellular machinery and increase production of interleukin-10 (IL-10)—an anti-inflammatory cytokine that suppresses immune response, allowing the bacteria to persist undetected. This mechanism may explain why patients experience cyclic flares and remissions, and why Bartonella can linger silently for years. Dr. Bush’s findings suggest that even short-term infections can produce measurable transcriptional changes in brain immune cells within 48 hours. If such infections persist for months or years, they may set the stage for neurodegenerative disease, particularly when combined with other pathogens or environmental factors. “If one intracellular pathogen can cause this many changes in two days, imagine what happens over months or years. Bartonella may be the spark that primes the brain for neurodegeneration.” — Dr. Janice Bush Why It Matters Dr. Bush’s research offers a groundbreaking look at how a common, underrecognized infection may drive neuroinflammation and neurodegeneration. Her work bridges veterinary medicine, infectious disease, and neurology—revealing how pathogens once dismissed as minor or self-limiting may alter the brain’s immune landscape. By demonstrating that Bartonella can infect and manipulate microglial cells, she provides critical biological evidence linking vector-borne disease and cognitive decline, paving the way for future diagnostic and therapeutic innovation. About the Event This interview was recorded at the 2nd Annual Alzheimer’s Pathobiome Initiative (AlzPI) and Philadelphia College of Osteopathic Medicine (PCOM) Symposium, held October 3, 2025, at Ohio University in Dublin, Ohio. The event gathered more than 20 leading researchers exploring how microbes, the microbiome, and immune dysregulation contribute to Alzheimer’s, dementia, and infection-associated chronic illness (IACI). The Tick Boot Camp Podcast, in partnership with Ali Moresco and Nikki Schultek, documented these conversations to connect the chronic Lyme, infectious disease, and Alzheimer’s research communities. This episode is part of Tick Boot Camp’s AlzPI collaboration series. Learn More Learn more about the Alzheimer’s Pathobiome Initiative (AlzPI)Listen to Tick Boot Camp Podcast episodes, including Episode 406: Pathobiome – An Interview with Nikki Schultek and Episode 101: The Young Gun – An Interview with Alex (Ali) Moresco discussed in this interview.
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    20 Min.
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