In this episode, Ben sits down with Mark Lintern to challenge that assumption and explore a radically different idea...

What if cancer is a disease of suppression, not failure?

Drawing on the metabolic theory, the Warburg effect, mitochondrial biology, and emerging research into the tumour microbiome, Mark lays out a compelling framework that reframes cancer as a chronic cell danger response potentially driven by persistent infection.

They unpack..

Why the metabolic theory explains more than genetics but still leaves gaps.

Why apoptosis and immune evasion don’t fully fit the “broken cell” narrative.

How mitochondria may intentionally suppress oxidative phosphorylation.

Why prostate cancer breaks the Warburg rule entirely.

The role of fungi, bacteria, and the tumour associated microbiome.

Why immunotherapy often fails and sometimes backfires.

How infection biology may explain inflammation, immune suppression, and tumour growth.

What this means for real world treatment strategies, from metabolic therapy to repurposed drugs.

This is a deep, systems-level conversation. Not about silver bullets but about first principles.

If theory informs treatment, then getting the theory right matters.

We cover:

🧬 Metabolic vs somatic mutation theory

🔥 The Warburg effect as a defensive response

🦠 Infection, fungi, and the tumour microbiome

🧠 The cell danger response model

🫁 Oxygen, mitochondria, and immune signalling

🧪 Prostate cancer as a metabolic outlier

💊 Repurposed drugs, antifungals, and metabolic therapies

🌱 Why cancer may be adaptive — not defective

This episode isn’t about certainty.

It’s about asking better questions.

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